Specificity, cross-reactivity, and function of antibodies elicited by Zika virus infection.

نویسندگان

  • Karin Stettler
  • Martina Beltramello
  • Diego A Espinosa
  • Victoria Graham
  • Antonino Cassotta
  • Siro Bianchi
  • Fabrizia Vanzetta
  • Andrea Minola
  • Stefano Jaconi
  • Federico Mele
  • Mathilde Foglierini
  • Mattia Pedotti
  • Luca Simonelli
  • Stuart Dowall
  • Barry Atkinson
  • Elena Percivalle
  • Cameron P Simmons
  • Luca Varani
  • Johannes Blum
  • Fausto Baldanti
  • Elisabetta Cameroni
  • Roger Hewson
  • Eva Harris
  • Antonio Lanzavecchia
  • Federica Sallusto
  • Davide Corti
چکیده

Zika virus (ZIKV), a mosquito-borne flavivirus with homology to Dengue virus (DENV), has become a public health emergency. By characterizing memory lymphocytes from ZIKV-infected patients, we dissected ZIKV-specific and DENV-cross-reactive immune responses. Antibodies to nonstructural protein 1 (NS1) were largely ZIKV-specific and were used to develop a serological diagnostic tool. In contrast, antibodies against E protein domain I/II (EDI/II) were cross-reactive and, although poorly neutralizing, potently enhanced ZIKV and DENV infection in vitro and lethally enhanced DENV disease in mice. Memory T cells against NS1 or E proteins were poorly cross-reactive, even in donors preexposed to DENV. The most potent neutralizing antibodies were ZIKV-specific and targeted EDIII or quaternary epitopes on infectious virus. An EDIII-specific antibody protected mice from lethal ZIKV infection, illustrating the potential for antibody-based therapy.

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عنوان ژورنال:
  • Science

دوره 353 6301  شماره 

صفحات  -

تاریخ انتشار 2016